By J. X. Thomas Jr. Ph.D., M. W. Gerdisch (auth.), Gerd Heusch, John Ross Jr. (eds.)
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Additional info for Adrenergic Mechanisms in Myocardial Ischemia
In: Usdin E, Kopin II, Barchas J (Eds) Catecholamines: Basic and Clinical Frontiers. Vol I, Pergamon Press, New York, Oxford, Toronto, Sydney, Frankfurt, Paris, pp268-270 46. Graefe KH, Zeitner CJ, Fuchs G, Keller B (1984) Role played by sodium in the membrane transport of 3H-noradrenaline across the axonal membrane of noradrenergic neurones. In: Fleming WW (Ed) Neuronal and Extraneuronal Events in Autonomic Pharmacology. Raven Press, New York, pp 51-62 47. Griffiths J, Leung F (1971) The sequential estimation of plasma catecholamines and whole blood histamine in myocardial infarction.
N Engl J Med 274: 1102-1108 139. Vatner DE (1989) Uncoupling of the p-adrenergic receptor by myocardial ischemia. In: Brachmann J, Schomig A (Eds) Adrenergic System and Ventricular Arrhythmias in Myocardial Infarction. Springer Verlag, NewYork-Berlin-Heidelberg, pp91-97 140. Vatner DE, Knight D, Shen YT, Thomas JX, Homcy CJ, Vatner SF (1988) One hour of myocardial ischemia in conscious dogs increases beta-adrenergic receptors, but decreases adenylate cyclase activity. J Mol Cell Cardiol 20: 75-82 141.
DaTorre Cardiovascular Division, Department of Internal Medicine and Department of Pharmacology, Washington University School of Medicine, st. Louis, Missouri, USA Summary The lXI-adrenergic receptor exists as at least two distinct subtypes, IX la and IXlb' Based on hydrophobic exclusion studies and limited proteolysis of the cloned receptor, it appears to possess characteristics analogous to other membrane-bound receptors including seven membrane spanning domains, three extracellular, and three intracellular loops, with extensive glycosylation near the extracellular amino terminus.
Adrenergic Mechanisms in Myocardial Ischemia by J. X. Thomas Jr. Ph.D., M. W. Gerdisch (auth.), Gerd Heusch, John Ross Jr. (eds.)