By Richard G. Cutler (auth.), John E. Johnson Jr. (eds.)
The getting older approach, like so much (all?) issues within the universe, is a puzzle. it truly is this type of primary prevalence, universal to all residing issues, that it satirically could be the most complex and confusing puzzle to resolve. There are, in fact, puzzles adequate in quantity to occupy all of the scientists and philosophers who've ever lived 1000 occasions over. but what different phenomenon impacts each person people up to ageing? interest concerning the ubiquity of getting older as a phenomenon, boosted possibly by means of an simply common worry of that very same phenomenon as an indisputable manifes tation of our personal mortality, has ended in the compilation of knowledge at the getting older procedure, that have gathered as speedily because the aged who could gain advantage from these info. many of the medical facts at the biology of getting older are on the biochemical and physiological degrees, whereas relatively little info has been avail capable on the anatomical point. due to this, a two-volume set known as getting older and mobile constitution was once conceived, the 1st quantity having been released in 1981 and the second one quantity being released at the same time with the current one on phone functionality. The emphasis on education of scientists and different contributors in complex schooling has, in the course of previous a long time, been considered one of elevated specialization. now not only will we concentrate on body structure, anatomy, zoology, or literature, yet in glomerular filtration, synaptic junctions, tree frogs of Brazil, or English poetry of the 19th century.
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3. Speciation during primate evolution is most closely related to changes occurring in the timing and degree of the same structural expression of genes found in all species, rather than in the appearance of new structural genes. 4. Longevity evolved at an extremely rapid rate during recent hominid evolution, resulting in the uniform extension of the length of time gen- 37 38 RICHARD G. CUTLER eral health is maintained. This result is most consistent with few genetic alterations being capable of decreasing the entire organismic complex of aging processes in a uniform and coordinate manner.
Man 96 man 0960's) 0800's) 84 72 ~ 60 >- a. 33 (ASM) + 4 • gibbon langur 12 tree shrew 2 6 4 10 8 14 12 16 18 Age at sexual maturation (yrs) Figure 10. Rate of development as a function of LSP in primate species. Data taken in part from Cutler (1976a,b; 1978). I. ~--------~ . 700 days "" .... 253 days .... .... 1000 days .... .... 953 days " I I 1138 days 138 days Figure 11. Relation between the amount of increased lifespan gained in rats on a restricted calorie diet in the presexual maturation period and the postsexual maturation periods of lifespan.
In this sense, then, the unusually slow developmental rate of the human is considered to be an essential mechanism accounting for his unusual longevity. Also, an important prediction of this concept is that any nontoxic method that decreases the overall rate of development would be expected to increase the species' LSP. This interpretation of the longevity-enhancing effects of lengthened developmental period may give us some additional insight into whether a calorierestricted diet might work in primate species as it clearly does in rodents.
Aging and Cell Function by Richard G. Cutler (auth.), John E. Johnson Jr. (eds.)